NotAIDS! Comment
by The Editor

And in point of fact, the dermatologists, when they looked back and began to see that there were a fair number of patients in their gay practices who had Kaposi's sarcoma but who ended up not having HIV. - Dr. John Ziegler

Remember PCP and KS, the scourge of gay men in cities across America? They were the face of AIDS. They WERE AIDS - later named HIV, said to be the underlying cause of AIDS.

What if they weren't AIDS? Would that beg the question, "What is AIDS anyway?"

Is it clear that what was referred to as PCP - an acronym for pneumocystis carinii pneumonia even related to AIDS? Kaposi's sarcoma, another early AIDS-defining illness, is now known to be NOT AIDS.

PCP, a fungal infection of the lungs, started as a small cluster of contagious infections under the tutelage of Los Angeles-based Dr. Gottlieb, who was in close touch with the Feds at CDC.

It eventually morphed first into GRID (gay-related immune deficiency) and then into "AIDS" in this concerted effort to see it not as any other cluster but as a homosexual cluster of immune dysfunction, as if therein lived the aetiology of the great and powerful mystery of gay men who fell ill.

No matter that almost every parasite harmful to humans causes immune dysfunction, and they are usually contagious.

As titillating as gay sex may be  read more »

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NotAIDS! News
December 30, 2008, Updated November 5, 2009

by The Editor

The death of alternative AIDS activist Christine Maggiore came as a shock to me, not only because for 17 years she'd offered herself as living proof that a so-called "HIV-positive" diagnosis was not necessarily fatal but, more importantly, because she'd been a close personal friend for 15 years. Her death does not change any of the scientific facts that make the idea that HIV is the sole cause of AIDS not only wrong, but ridiculous; though supporters of the mainstream view of AIDS are exploiting it for propaganda, what it really underscores is the need for more basic research to document whether "HIV-positive" people who take the highly toxic AIDS medications actually live longer, healthier lives than those who don't. - Mark Gabrish, Conlan/Zenger's Newsmagazine^*

When the rabid AIDS promoters hit the media channels and the Internet gloating over the death of leading HIV "rethinker" Christine Maggiore, it will be in the sadistic manner of which only AIDS-lovers are capable, and they will twist the truth into a scary fable meant to coax you into taking your meds.

Yet, it would be prudent to resist any temptation to believe their false assumption that Christine Maggiore succumbed to an "AIDS-related" illness, specifically, HIV-related pneumonia. If she tested negative, after testing positive, and her son tests negative, how does her death from bacterial pneumonia prove she died from HIV infection? Even if she was HIV positive, on what basis is her death attributed to HIV?

In an international study of bacterial pneumonia outcomes, conducted in part by the University of Alberta, researchers concluded that pneumonia doesn't appear to harm HIV-positive patients any more than those who are HIV-negative.

There was also negligible difference in the mortality rate; total deaths among the HIV patients was 3.5 per cent (two of 58 patients), and 4.8 per cent (seven of 174) among the HIV-negative patients.

Mourners and revelers alike, consider the following, as noted on Medicine.net.

Currently, over 3 million people develop pneumonia each year in the United States. Over a half a million of these people are admitted to a hospital for treatment. Although most of these people recover, approximately 5% will die from pneumonia. Pneumonia is the sixth leading cause of death in the United States.

The latest numbers from the CDC put pneumonia as the 8th leading cause of death in the United States as of the last officially available government numbers from 2005.

 read more »

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It was over a year of symptoms increasing in severity, and more disabling with time, that culminated in profoundly depleted platelets. Platelets are the elements in blood that clot to prevent bleeding uncontrollably, either in the body or on the skin when cut.

My neck was stiff, and felt like muscles were twisted around a nerve, shooting electrial pulses of pain out to my shoulder. My lower back felt as if the cartilage was disintegrating and the lowest disks would grind and snap. It was difficult to stand up straight or walk if I dared to bend my back at all to pick something up off the floor.

My knee became arthritic, as did my left thumb joint which made gripping anything impossible.

But it was the second blood test that showed my platelets were dangerously low, and increasing difficulty walking that led me to take control of my own care, and figure out what was wrong with me. My ankles became swollen and painful, my and shins, (tibia) felt like someone had taken a sledgehammer to them. They were covered with red, hot patches that were strangely reminiscent of the migrating rash I had after the curious black blister over a year earlier.

Iin the absence of any medical staff I diagnosed myself finally, after 10 months of multiplying symptoms. I found a picture online of a blood engorged tick- it looked identical to the black blister I had taken a heat gun to last August because of the intense (there is no word adequate to describe it) itch that was deep below that blister. I had then washed the blister off with my hand and soap down the shower drain.

After the allergic reaction thatlasted 3 weeks with intense itching and microblistering, and oozing, the site of the blister finally healed and suddenly within days, a hot red blotchy patch with clearly demarcated, boundaries appeared under my arm ( I can't remember now if it was the left or right), and then disappeared quickly, and within hours, it reappeared on my neck. It felt like I had been branded with a hot iron. I believe this was the "migrating rash" that is described on many lyme disease web sites.

When my symptoms climaxed after months of feeling like I had morphed into an arthritic and crustry old man (I'm a young 39 who has never had any symptoms of back pain or any joint pain) I had to go to an emergency room armed with research, the photo of the engorged tick I found online, and specifically ask for a Doxycyclene prescription.

Most Doctors didn't believe i had been bitten by a tick or thougt I was being delusional and 3 docotrs at the local gay clinic told me it was HIV related, and even told me the low platelets were an HIV condition and if I only went on meds it would resolve. This is ludicrous as I told these doctors about my experience with a black blister that I think may have been a tick feasting on my stomach.  read more »

NotAIDS! Blog
July 4, 2009

HIV gets blamed a lot. From anemia to dementia, wasting, lymphomas, and fungal infections, it is also blamed for TB, pneumonia, KS, "wasting", obesity, and yes, even car accidents in some countries.

I recently had a strange migrating red rash on my shins, and ankles. The red patchy areas were hot and my ankles and calves, and shins were extremely tender, painful, and felt like they were atrophying.

I went to the clinic where I get my labs, saw a doctor who said I injured myself, did I maybe go on a drinking or drug binge and not remember what happened. I was sent on my way with a grin and a nod.

I had my labs done a few days later, 5 or whatever tubes of blood were taken.

My legs seemed like they were healing, at least the red hot patches were going away.

The HIV doctor at the clinic where I have my labs done called me on a Tuesday morning. There was an abnormal result on my labs, did I have time to talk for a few.

He said I had 25,000 platelets per microliter. Under 20,000 you can have dangerous nosebleeds and under 10,000 you can have a spontaneous brain or stomach bleed - and die.

Doc told me to make an appointment right away to start AIDS drugs, and that should take care of my low platelets. I asked about other more specific causes and he couldn't name one. My appointment day I declined the Prednisone, and declined the HIV drugs. I would wait 10 days and retest and then make my decision.

I read a paper about low platelets - thrombocytopenia - and they said this condition frequently occurs in HIV+ people. Then it quoted a study where only 3% of a random HIV+ cohort had thrombocytopenia. Most studies show a prevalence of about 20% of all HIV+ populations with clinically insignificant low platelet counts (higher than 20,000).

My research showed about 140 causes of thrombocytopenia. Here are just a few items that caught my attention.  read more »

by Ulrich E. Schaible*, Stefan H. E. Kaufmann

I am not interested in the bloody system! Why has he no food? Why is he starving to death?                                          - Bob Geldof in The Observer, 1985

Activation and sustenance of immune responses during infection requires increased energy consumption. Protein energy malnutrition (PEM) is a critical, yet underestimated factor in susceptibility to infection, including the “big three” infectious diseases: HIV/AIDS, tuberculosis, and malaria. In this article, we discuss current concepts and controversies surrounding the complex influences of malnutrition on infection and immunity, and point to practical consequences of countermeasures in acute malnutrition.

We call for new strategies to overcome worldwide morbidity and mortality caused by chronic malnutrition in impoverished countries and by the newly emerging public health threat of overnutrition in industrialized societies.

Background 

In response to infection, the immune system first executes innate and then subsequently acquired host defense functions of high diversity. Both processes involve activation and propagation of immune cells and synthesis of an array of molecules requiring DNA replication, RNA expression, and protein synthesis and secretion, and therefore consume additional anabolic energy. Mediators of inflammation further increase the catabolic response. Studies in a simple system, involving measurement of the survival of malnourished bumblebee workers, showed that the energy cost of immunity further impairs fitness [1].

Consequently, the nutritive status of the host critically determines the outcome of infection.

Apart from deficiencies in single nutrients, such as vitamins, fatty acids, amino acids, iron, and trace elements, undernourishment based on PEM greatly increases susceptibility to major human infectious diseases in low-income countries, particularly in children [2–4].

Malnutrition is responsible, directly or indirectly, for 54% of the 10.8 million deaths per year in children under five and contributes to every second death (53%) associated with infectious diseases among children under five years of age in developing countries [5]. Infection causes energy loss on the part of the individual, which reduces productivity on the community level and perpetuates the alarming spiral of malnutrition, infection, disease, and poverty (Figure 1).  read more »