No difference between HIV positive and negative?

NotAIDS! Essay
May 13, 2007 (updated)

There's not a soul on this planet who hasn't heard of HIV, and most of these people have probably heard of the mechanism by which "they" say the virus stalks and kills its prey. Laying in wait for an opportune time, a myriad of infectious diseases emerge, ready to invade organs, tissues, and even the brain of the host.

10, 20, or 30 years after contacting HIV, this army of illnesses available to homo sapiens supposedly springs into action, ravaging the human host and eventually causing a fetid, emaciated death.

As the special people who inhabit the AIDS world have pointed out 1, it doesn't matter if the deadly infections are caused directly by the skillful labor of T-cell gobbling human immunodeficiency virions, or indirectly from the "reconstituted" immune system.

For the uninitiated, virions are particles of DNA or RNA which contain instructions for replication via protein synthesis. A virus is not a complete cell so it isn't ever a living organism.

In all species of virus that affect human health, the immune system reacts to a viral invader by sending their own armies of white blood cells, CD4 and CD8 cells and other macrophages.

We feel ill when the inflammation of tissue or the accumulation of waste from this process reaches critical mass. Then, as the white blood cells dump the whole mess to be excreted in our urine and the gases via our exhaled breath, we begin to feel better.

Now 'educated' to the marauding invader's identity, these macrophages, CD4 and CD8 cells establish future immunity by blocking the virus' activity before an inflamed response has a chance to make us feel ill.

HIV is different, the AIDS people persistently assert. By itself HIV will not cause illness; rather it is an immune system-specific molecule whose sole purpose is to kill off the CD4 cells.

According to their logic, AIDS, a very bad thing you get from a very bad activity - sex - is caused by HIV's assault on the immune system. It is characterized by other bacterial and fungal infections like tuberculosis, pneumnonia, and thrush, an overgrowth of yeast found naturally in all humans. They even say it causes viral infections like cervical cancer, and lymphomas.

However, if this were true, wouldn't there be significant differences between HIV negatives and HIV positives in how serious infections such as pneumonia, candida (thrush), and tuberculosis (TB) resolve?

At the 1998 12th World AIDS Conference in Geneva, Switzerland, the following observation was made in a report on TB in South African miners:

Record Rates of TB in South African Gold Miners

The 1995 tuberculosis rate at 4 gold mines near Johannesburg was 1318/100,000. The 376 culture positive cases from 1995 were treated through health care facilities at the miners' hostels. In spite of an HIV incidence of 49% among the TB cases, the TB cure rates were 89% for HIV-seropositives and 88% for HIV seronegatives.

Study after study shows that there is really no difference between HIV negatives and HIV seropositives who are outside the AZT-ARV-ART-HAART sphere, meaning they are treatment naive, or pharm-free.

Therefore, HIV negatives, who who don't test very frequently, and HIV positive souls who brave the anger of AIDS-world experts by not taking the meds, get the same illnesses, react the same to treatments which target the illness (unlike antiretrovirals, which are not specific to any one viral particle or bacterial cell), and survive with the same mental acuity.

Going back as far to the early days, a report on the 3-year Multicenter AIDS Cohort Study (MACS), delivered at the 1989 International Conference on AIDS, established a similar conclusion.

"We observed no evidence of gradual decline on this measure among otherwise asymptomatic seropositive subjects. These data support previous cross-sectional data from the MACS suggesting no increased risk for cognitive abnormalities among asymptomatic seropositive individuals."2

In a recent study of 174 HIV negatives and 58 HIV positives, age and gender matched, no differences were found in the patients' stabilization and recovery. Nor were there differences in morbidity; indeed the HIV negative cohort had a slightly higher death rate:

There was also negligible difference in the mortality rate; total deaths among the HIV patients was 3.5 per cent (two of 58 patients), and 4.8 per cent (seven of 174) among the HIV-negative patients.

A 2007 study comparing Candida infection response to the promising new anti-fungal agent, micafungin (FK463)3 and the widely prescribed amphotericin B, showed that the "effectiveness of either treatment was independent of primary site of infection and the patient's immune status."4

If you are HIV negative, and you, like the other millions on this planet, have one of these afflictions, you better be prepared to explain yourself. In some parts of the world, it doesnt' matter if you test negative, they'll say you have AIDS, and shove meds down your throat until you get the other 29 infections.

Death will also occur from the new & improved immune system, super-engineered and hyper-manipulated by AZT, protease and integrase inhibitors, and the entire extended family of radioactive and mysterious AIDS drugs.

IRIS, "immune reconstitution inflammation syndrome" is what happens when the 'medicine' worked too well,1 and they are exactly the same diseases they call AIDS.

If you are HIV positive and don't take the meds you'll be blessed with a miraculous sounding label: long term non-progressors, or "elite controllers." If you take the meds, they'll say it was IRIS.
Just die please seems to be the message. If it isn't happening soon enough, they "can do things" to speed things up.

As long as their subjects die, AIDS researchers seem very happy, keeping busy with papers and conferences, and they are perfectly gleeful to receive lots of support from the world's leading pharmaceutical companies.

Do not be surprised if you find yourself wondering whether the only difference to be acknowledged is between the victims of DNA altering AIDS drugs and the non-victims.

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1. Read NotAIDS! Investigative Report.

2. Changes in performance on the trail-making test before and after HIV-1 seroconversion and diagnosis of AIDS: the Multicenter AIDS Cohort Study (MACS). Miller EN, Selnes OA, Visscher B, McArthur JC, Satz P, Dudley J. Int Conf AIDS 1989 Jun 4-9; 5: 464 (abstract no. Th.B.P.288). University of California, Los Angeles, CA, USA

3. Pure and Applied Chemistry, 2007, Volume 79, No. 4, pp. 603-614. Discovery of micafungin (FK463): A novel antifungal drug derived from a natural product lead. Akihiko Fujie; Fermentation Research Laboratories, Astellas Pharma Inc., 5-2-6 Tokodai, Tsukuba, Ibaraki 300-2698, Japan

4. New Treatment For Fungi Which Cause Thrush And Sepsis; Medical News Today Online using source: The Lancet. Accessed May 11, 2007.

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